Hormesis: A Revolution in Biology, Toxicology and MedicineHormesis is a poorly understood phenomenon affecting all forms of life on earth. This groundbreaking book summarizes and analyzes the various positives of hormesis in an attempt to reveal hormesis as a fundamental principle of biomedical sciences as a whole. |
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Inhalt
1 | |
References | 11 |
Once Marginalized Evidence Now Supports Hormesis as the Most Fundamental Dose Response | 15 |
References | 51 |
The Fundamental Role of Hormesis in Evolution | 57 |
References | 66 |
Transcriptional Mediators of Cellular Hormesis | 69 |
References | 87 |
References | 119 |
Dietary Energy Intake Hormesis and Health | 123 |
References | 133 |
The Antithesis of Hormesis | 139 |
References | 147 |
Hormesis and Aging | 153 |
References | 169 |
The Future of Natural Productsand ManMade Drugs in Disease Prevention and Treatment | 177 |
Complexity of Receptor Systemsand Responses | 94 |
References | 105 |
ExerciseInduced Hormesis | 109 |
References | 195 |
199 | |
Andere Ausgaben - Alle anzeigen
Hormesis: A Revolution in Biology, Toxicology and Medicine Mark P. Mattson,Edward J. Calabrese Keine Leseprobe verfügbar - 2010 |
Hormesis: A Revolution in Biology, Toxicology and Medicine Mark P. Mattson,Edward J. Calabrese Keine Leseprobe verfügbar - 2014 |
Hormesis: A Revolution in Biology, Toxicology and Medicine Mark P. Mattson,Edward J. Calabrese Keine Leseprobe verfügbar - 2009 |
Häufige Begriffe und Wortgruppen
action activity adaptive addition agents aging animal antioxidant assessment beneficial effects binding Biol biological biphasic brain Calabrese caloric restriction cancer cells cellular Chem chemical complex concept couch potato damage dietary disease dose dose-response relationship drugs effects energy enhanced environmental et al example exercise exposure expression FOXO function genes glucose growth heat shock hormesis hormetic hormetic dose response hormetic effects human important improve increased induce insulin interaction involved kinase levels lifespan longevity low doses low-dose Mattson MP mechanisms mediate mice mild moderate molecular multiple muscle nature neurons Nrf2 observed occur organisms oxidative stress pathways performance Pharmacol Physiol potential production proliferation promotes protect proteins range rats Rattan receptor reduced regulation reported resistance result risk role running shown signaling specific stimulation stress response stressors studies threshold tissue toxic Toxicol transcription factors treatment various zone