Chemical Brain InjuryVan Nostrand Reinhold, 1998 - 392 Seiten chemical brain injury Kaye H. Kilburn, M.D. The idea that the brain is the most susceptible body organ to the adverse effects of chemicals seemed a remote possibility a decade or two ago. Among the skeptics was Dr. Kaye H. Kilburn. Well-known in Environmental Medicine and Occupational Health, he had demonstrated that airways-obstruction caused the Monday-morning asthma from cotton dust in textile workers that led to the Cotton Dust Standard. He showed how asbestos scarred the lungs small airways to trap air and reduce vital capacity, and that welding fumes, aluminum refining, diesel exhaust, and formaldehyde caused asthma by narrowing small airways. These workers complaints of memory loss, inability to concentrate, dizziness, lightheadedness, and loss of balance led Dr. Kilburn to consider how to measure brain functions. He borrowed and adapted tests and built devices to measure key brain activities, and learned to find out how symptoms predicted losses of balance, quickness, and strength, and loss of vision for color and form. Nearly 300 patients who had been exposed to chemicals were evaluated for diagnosis and nearly 4,000 people who had been exposed in groups were evaluated for chemical effects. Statistical analysis of patients gathered from individuals and groups in cities, towns, and rural areas provided complementary insights into the effects of chemicals. Chemical Brain Injury focuses on how common and abundant chemicals affect the brain. It synthesizes endeavors to assess the effects of chemicals that were gathered over 15 years and published in 30 widely-scattered papers. Included are effects of chlorine, hydrogen sulfide, chlordane, arsenic, trichoroethylene, PCBs, hydrochloric acid, and diesel exhaust. To understand how individuals and populations respond to chemicals, it considers the effects of aging, of years of school completed, and of sex, height, weight, and other factors. The book addresses such issues as: The infrequency of chemically unaffected populations in the United States Mechanisms of brain damage from chemicals Prognosis and therapy The last section examines the social context and discusses such matters as: The future of neurotoxicology, including needs and responsibility Legal proceedings Social changes Chemical Brain Injury is the definitive volume on the adverse effects of chemical exposure on the human brain. |
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Seite xv
... seemed exaggerated and overly dra- matic . Carson's plea for stronger regulatory guidelines for pesticides was thought to have overlooked " the considerable tightening that's taken place in the past few years , ' commented Dexter ...
... seemed exaggerated and overly dra- matic . Carson's plea for stronger regulatory guidelines for pesticides was thought to have overlooked " the considerable tightening that's taken place in the past few years , ' commented Dexter ...
Seite 71
... seemed a plausible expecta- tion . Analysis was started with 10 to 20 patients exposed to hydrogen sulfide , chlordane , chlorine , diesel exhaust - aldehydes , and formaldehyde ( Table 4.4 ; Figures 4.2a - d ) . Ten- tative patterns ...
... seemed a plausible expecta- tion . Analysis was started with 10 to 20 patients exposed to hydrogen sulfide , chlordane , chlorine , diesel exhaust - aldehydes , and formaldehyde ( Table 4.4 ; Figures 4.2a - d ) . Ten- tative patterns ...
Seite 308
... seemed to affect some tests in the same way as did chemicals . The implication of these studies is that chemicals adversely affected 9 of 13 , just over two - thirds , of the control groups . Although this conclusion may surprise ...
... seemed to affect some tests in the same way as did chemicals . The implication of these studies is that chemicals adversely affected 9 of 13 , just over two - thirds , of the control groups . Although this conclusion may surprise ...
Inhalt
Neurobehavioral Effects and Exposure Epidemiology | 6 |
Deriving Prediction | 21 |
Neuropsychological Tests | 29 |
Urheberrecht | |
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Häufige Begriffe und Wortgruppen
abnormal adults by ANOVA aluminum ANOVA arsenic astrocytes balance bars are statistically blink reflex latency block design brain damage central nervous system chemical exposure chlordane chlorine choice reaction clinical coefficients compared to unexposed Comparison Culture Fair diesel Digit Symbol disease disorders effects environmental exposed and unexposed exposed subjects eyes closed Eyes Open FIGURE Finger Writing formaldehyde Hatched bars hydrogen sulfide irritation KH and Warshaw Kilburn Kilburn KH limbic system lupus erythematosus Mean Sd Mean measured memory loss Muscle Shoals neurobehavioral neurological neurophysiological neuropsychological neurotoxic occupational patients PCBs Pegboard Perceptual Motor Speed Picture Completion POMS score populations predicted profile of mood pulmonary refinery Sd Mean Sd seizures shortness of breath showed Simple Reaction solvents statistically significant Supraorbital Tap sway speed symptom frequencies Table Temporal lobe test scores toluene toxic Trails trichloroethylene unexposed adults unexposed group verbal recall vinyl chloride visual fields Vocabulary Warshaw RH Wickenburg women workers
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