Hormesis: A Revolution in Biology, Toxicology and MedicineMark P. Mattson, Edward J. Calabrese Springer Science & Business Media, 01.12.2009 - 213 Seiten Hormesis is a poorly understood phenomenon affecting all forms of life on earth. This groundbreaking book summarizes and analyzes the various positives of hormesis in an attempt to reveal hormesis as a fundamental principle of biomedical sciences as a whole. |
Inhalt
1 | |
11 | |
Once Marginalized Evidence Now Supports Hormesis as the Most Fundamental Dose Response | 15 |
References | 51 |
The Fundamental Role of Hormesis in Evolution | 57 |
References | 66 |
Transcriptional Mediators of Cellular Hormesis | 69 |
References | 87 |
References | 119 |
Dietary Energy Intake Hormesis and Health | 123 |
References | 133 |
The Antithesis of Hormesis | 139 |
References | 147 |
Hormesis and Aging | 152 |
References | 169 |
The Future of Natural Productsand ManMade Drugs in Disease Prevention and Treatment | 177 |
Complexity of Receptor Systemsand Responses | 94 |
References | 105 |
ExerciseInduced Hormesis | 109 |
References | 195 |
199 | |
Andere Ausgaben - Alle anzeigen
Hormesis: A Revolution in Biology, Toxicology and Medicine Mark P. Mattson,Edward J. Calabrese Keine Leseprobe verfügbar - 2010 |
Hormesis: A Revolution in Biology, Toxicology and Medicine Mark P. Mattson,Edward J. Calabrese Keine Leseprobe verfügbar - 2014 |
Hormesis: A Revolution in Biology, Toxicology and Medicine Mark P. Mattson,Edward J. Calabrese Keine Leseprobe verfügbar - 2009 |
Häufige Begriffe und Wortgruppen
Acad Sci USA activity agents Alzheimer’s disease antioxidant Appl Physiol assessment BDNF beneficial effects binding Biogerontology Biol Chem biological biphasic dose responses brain Ca2+ Calabrese Calabrese and Baldwin caloric restriction cancer cells cellular chemical Crit Rev Toxicol damage diabetes dose-response model dose-response relationship drugs effects of exercise enhance environmental fibroblasts FOXO free radicals function G protein-coupled receptor glucose GPCR growth factors heat shock heat shock factor heat-shock proteins hippocampus homeodynamic hormesis hormetic dose response hormetic effects hormetic responses HSF1 human increase induce insulin interaction levels lifespan ligand longevity low doses low-dose Mattson MP Maudsley mediate metabolic mice mild stress moderate exercise molecular Natl Acad Sci neurons Neurosci neurotrophic factor NF-kB Nrf2 organisms oxidative stress Pharmacol phosphorylation phytochemicals Proc Natl Acad proliferation proteasomal protect protein-coupled rats Rattan SIS receptor regulation Resveratrol role signaling pathways stimulation stressors studies survival threshold tissue toxic transcription factors